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Atherosclerosis 25 July 2009

Atherosclerosis is NOT a disease of high cholesterol. Atherosclerosis is a disease of overly acidic blood, fungal mycotoxins, heavy metals and free radicals which injure the inner arterial wall (endothelium). The sequence of events is as follows:

  1. Fungal mycotoxins and free radicals produced by acidic blood, heavy metals, insulin, iron and homocysteine inflame (oxidise) the arterial wall. This process is known as oxidative stress.
  2. Cholesterol is attracted to the inflamed arterial wall in an attempt to repair the damage.
  3. The cholesterol is then attacked by the free radicals and oxidised (becomes rusty).
  4. The “rusty” cholesterol is then consumed by white cells.
  5. The white cells (known as foam cells) then burst and release more inflammatory mediators which inflame the arterial wall more.
  6. Acid blood and free radicals also increase the amount of free calcium in the blood which is then attracted to the inflamed arterial wall in an attempt to “glue” the cracks together.
  7. The arterial wall, in a vicious cycle, is left with ever increasing amounts of inflammation, build up of oxidised cholesterol and the calcium glue. Increasing research also shows that these plaques have high levels of pathogenic organisms. A study published in the November 2006 edition of the Journal of Vascular Surgery found that multiple pathogenic organisms were involved in the walls of aortic aneurysms.
  8. This produces plaques in the wall of the artery, narrowing the artery and reducing blood and oxygen flow to tissues.
  9. As the plaque increases in size, it eventually becomes unstable and ruptures.
  10. A blood clot forms at the site of the rupture, completely blocking the artery. If the artery supplies the heart, a heart attack occurs. If it supplies the brain, a stroke occurs.

The problems on just focusing on cholesterol:

  • The root causes of the inflammation of the arterial wall is ignored.
  • All fat has been implicated as causing a problem. Many fats are healthy to eat. Hydrogenated fats and trans fatty acids that are present in commercially available vegetable oils and processed foods are the real culprits because they increase free radicals.

Arteries are much healthier if the cause of the inflammation is addressed:

  • Free radicals/oxidative stress
  • Chronic fungal infections. Professor AV Costantini wrote in his book Fungalbionics that “there is a known cause of atherosclerosis and that cause is the fungi and their mycotoxins.”
  • Excessive acidity.
  • Excessive free calcium.
  • Chronic infections
  • Heavy metals such as lead and mercury.

There are several tests which give a much better idea of the risks of atherosclerosis than just doing a cholesterol level. These include:

  • Homocysteine- inflames the arterial wall.
  • Fibrinogen- a clotting factor that accumulates at the site of damaged enothelium.
  • Glucose- high levels stiffen and damage the arterial wall.
  • Ferritin- high iron stores oxidise the LDL.
  • Testosterone- low levels interfere with normal endothelial function.
  • Vitamin K- low levels enable calcium to be deposited in the arterial wall rather than bone.
  • C-reactive protein- an inflammatory marker from the liver which also directly damages the endothelium.
  • Lipoprotein A- oxidises LDL.
  • Vitamin D.

To avoid atherosclerosis, follow my easy 10 point plan:

  1. Reduce heavy metals and other environmental toxins.
  2. Reduce pathogenic organisms
  3. Keep homocysteine down and vitamin K2 up
  4. Reduce inflammation by taking pharmaceutical grade fish oil.
  5. Reduce toxins by exercising daily and having regular saunas.
  6. Get 20-30 minutes of sun/day.
  7. Maintain physiological levels of balanced hormones.
  8. Reduce free radical levels by taking Coenzyme 10 daily.

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